Acoustic tweezers for the life sciences
Ozcelik, Adem, Joseph Rufo, Feng Guo, Yuyang Gu, Peng Li, James Lata, and Tony Jun Huang. 2018. “Acoustic Tweezers for the Life Sciences”. Nature Methods 15 (12): 1021–1028.
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Genomic Copy Number Dictates a Gene-Independent Cell Response to CRISPR/Cas9 Targeting
Aguirre, A. J., R. M. Meyers, B. A. Weir, F. Vazquez, C. Z. Zhang, U. Ben-David, A. Cook, et al. 2016. “Genomic Copy Number Dictates a Gene-Independent Cell Response to CRISPR/Cas9 Targeting”. Cancer Discov 6: 914-29.
Amplification of human papillomavirus genomes in vitro is dependent on epithelial differentiation
Bedell, M. A., J. B. Hudson, T. R. Golub, M. E. Turyk, M. Hosken, G. D. Wilbanks, and L. A. Laimins. 1991. “Amplification of Human Papillomavirus Genomes in Vitro Is Dependent on Epithelial Differentiation”. J Virol 65: 2254-60.
The landscape of chromosomal aberrations in breast cancer mouse models reveals driver-specific routes to tumorigenesis
Ben-David, U., G. Ha, P. Khadka, X. Jin, B. Wong, L. Franke, and T. R. Golub. 2016. “The Landscape of Chromosomal Aberrations in Breast Cancer Mouse Models Reveals Driver-Specific Routes to Tumorigenesis”. Nat Commun 7: 12160.
Genetic and transcriptional evolution alters cancer cell line drug response
Ben-David, U., B. Siranosian, G. Ha, H. Tang, Y. Oren, K. Hinohara, C. A. Strathdee, et al. 2018. “Genetic and Transcriptional Evolution Alters Cancer Cell Line Drug Response”. Nature 560: 325-30.
An ecosystem of cancer cell line factories to support a cancer dependency map
Boehm, J. S., and T. R. Golub. 2015. “An Ecosystem of Cancer Cell Line Factories to Support a Cancer Dependency Map”. Nat Rev Genet 16: 373-4.
Identification of distinct molecular phenotypes in acute megakaryoblastic leukemia by gene expression profiling
Bourquin, J. P., A. Subramanian, C. Langebrake, D. Reinhardt, O. Bernard, P. Ballerini, A. Baruchel, et al. 2006. “Identification of Distinct Molecular Phenotypes in Acute Megakaryoblastic Leukemia by Gene Expression Profiling”. Proc Natl Acad Sci U S A 103: 3339-44.
Chemical genetic strategy identifies histone deacetylase 1 (HDAC1) and HDAC2 as therapeutic targets in sickle cell disease
Bradner, J. E., R. Mak, S. K. Tanguturi, R. Mazitschek, S. J. Haggarty, K. Ross, C. Y. Chang, et al. 2010. “Chemical Genetic Strategy Identifies Histone Deacetylase 1 (HDAC1) and HDAC2 As Therapeutic Targets in Sickle Cell Disease”. Proc Natl Acad Sci U S A 107: 12617-22.
Amplification of CRKL induces transformation and epidermal growth factor receptor inhibitor resistance in human non-small cell lung cancers
Cheung, H. W., J. Du, J. S. Boehm, F. He, B. A. Weir, X. Wang, M. Butaney, et al. 2011. “Amplification of CRKL Induces Transformation and Epidermal Growth Factor Receptor Inhibitor Resistance in Human Non-Small Cell Lung Cancers”. Cancer Discov 1: 608-25.
Expression analysis with oligonucleotide microarrays reveals that MYC regulates genes involved in growth, cell cycle, signaling, and adhesion
Coller, H. A., C. Grandori, P. Tamayo, T. Colbert, E. S. Lander, R. N. Eisenman, and T. R. Golub. 2000. “Expression Analysis With Oligonucleotide Microarrays Reveals That MYC Regulates Genes Involved in Growth, Cell Cycle, Signaling, and Adhesion”. Proc Natl Acad Sci U S A 97: 3260-5.