LC3s hire membrane breakers to attack viral shelters.
Biering, Scott B, Jayoung Choi, Hailey M Brown, and Seungmin Hwang. 2017. “LC3s Hire Membrane Breakers to Attack Viral Shelters.”. Autophagy 13 (11): 2010-12.
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).
Klionsky, Daniel J, Kotb Abdelmohsen, Akihisa Abe, Md Joynal Abedin, Hagai Abeliovich, Abraham Acevedo Arozena, Hiroaki Adachi, et al. 2016. “Guidelines for the Use and Interpretation of Assays for Monitoring Autophagy (3rd Edition).”. Autophagy 12 (1): 1-222.
Metabolic dysfunction drives a mechanistically distinct proinflammatory phenotype in adipose tissue macrophages.
Kratz, Mario, Brittney R Coats, Katherine B Hisert, Derek Hagman, Vesco Mutskov, Eduard Peris, Kelly Q Schoenfelt, et al. 2014. “Metabolic Dysfunction Drives a Mechanistically Distinct Proinflammatory Phenotype in Adipose Tissue Macrophages.”. Cell Metabolism 20 (4): 614-25.
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https://www.ncbi.nlm.nih.gov/myncbi/seungmin.hwang.1/bibliography/public/
CD300LF Polymorphisms of Inbred Mouse Strains Confer Resistance to Murine Norovirus Infection in a Cell Type-Dependent Manner.
Furlong, Kevin, Scott B Biering, Jayoung Choi, Craig B Wilen, Robert C Orchard, Christiane E Wobus, Christopher A Nelson, et al. 2020. “CD300LF Polymorphisms of Inbred Mouse Strains Confer Resistance to Murine Norovirus Infection in a Cell Type-Dependent Manner.”. Journal of Virology 94 (17).
Murine Norovirus Infection Induces TH1 Inflammatory Responses to Dietary Antigens.
Bouziat, Romain, Scott B Biering, Elaine Kouame, Kishan A Sangani, Soowon Kang, Jordan D Ernest, Mukund Varma, et al. 2018. “Murine Norovirus Infection Induces TH1 Inflammatory Responses to Dietary Antigens.”. Cell Host & Microbe 24 (5): 677-688.e5.
LysMD3 is a type II membrane protein without an in vivo role in the response to a range of pathogens.
Yokoyama, Christine C, Megan T Baldridge, Daisy W Leung, Guoyan Zhao, Chandni Desai, Ta-Chiang Liu, Vladimir E Diaz-Ochoa, et al. 2018. “LysMD3 Is a Type II Membrane Protein Without an in Vivo Role in the Response to a Range of Pathogens.”. The Journal of Biological Chemistry 293 (16): 6022-38.
Quo vadis? Interferon-inducible GTPases go to their target membranes via the LC3-conjugation system of autophagy.
Choi, Jayoung, Scott B Biering, and Seungmin Hwang. 2017. “Quo Vadis? Interferon-Inducible GTPases Go to Their Target Membranes via the LC3-Conjugation System of Autophagy.”. Small GTPases 8 (4): 199-207.
Targeting by AutophaGy proteins (TAG): Targeting of IFNG-inducible GTPases to membranes by the LC3 conjugation system of autophagy.
Park, Sungwoo, Jayoung Choi, Scott B Biering, Erin Dominici, Lelia E Williams, and Seungmin Hwang. 2016. “Targeting by AutophaGy Proteins (TAG): Targeting of IFNG-Inducible GTPases to Membranes by the LC3 Conjugation System of Autophagy.”. Autophagy 12 (7): 1153-67.
Gamma-herpesvirus kinase actively initiates a DNA damage response by inducing phosphorylation of H2AX to foster viral replication.
Tarakanova, Vera L, Van Leung-Pineda, Seungmin Hwang, Chiao-Wen Yang, Katie Matatall, Mickael Basson, Ren Sun, Helen Piwnica-Worms, Barry P Sleckman, and Herbert W Virgin. 2007. “Gamma-Herpesvirus Kinase Actively Initiates a DNA Damage Response by Inducing Phosphorylation of H2AX to Foster Viral Replication.”. Cell Host & Microbe 1 (4): 275-86.