LTD expression is independent of glutamate receptor subtype.
Granger, Adam J, and Roger A Nicoll. 2014. “LTD Expression Is Independent of Glutamate Receptor Subtype.”. Frontiers in Synaptic Neuroscience 6: 15.
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Genetic analysis of neuronal ionotropic glutamate receptor subunits.
Granger, Adam J, John A Gray, Wei Lu, and Roger A Nicoll. 2011. “Genetic Analysis of Neuronal Ionotropic Glutamate Receptor Subunits.”. The Journal of Physiology 589 (17): 4095-101.
Potentiation of synaptic AMPA receptors induced by the deletion of NMDA receptors requires the GluA2 subunit.
Lu, Wei, John A Gray, Adam J Granger, Matthew J During, and Roger A Nicoll. 2011. “Potentiation of Synaptic AMPA Receptors Induced by the Deletion of NMDA Receptors Requires the GluA2 Subunit.”. Journal of Neurophysiology 105 (2): 923-8.
Gefitinib induces myeloid differentiation of acute myeloid leukemia
Stegmaier, K., S. M. Corsello, K. N. Ross, J. S. Wong, D. J. DeAngelo, and T. R. Golub. 2005. “Gefitinib Induces Myeloid Differentiation of Acute Myeloid Leukemia”. Blood 106: 2841-8.
Corrigendum: The genomic landscape of juvenile myelomonocytic leukemia
Stieglitz, E., A. N. Taylor-Weiner, T. Y. Chang, L. C. Gelston, Y. D. Wang, T. Mazor, E. Esquivel, et al. 2016. “Corrigendum: The Genomic Landscape of Juvenile Myelomonocytic Leukemia”. Nat Genet 48: 101.
Association of Cell-Free DNA Tumor Fraction and Somatic Copy Number Alterations With Survival in Metastatic Triple-Negative Breast Cancer
Stover, D. G., H. A. Parsons, G. Ha, S. S. Freeman, W. T. Barry, H. Guo, A. D. Choudhury, et al. 2018. “Association of Cell-Free DNA Tumor Fraction and Somatic Copy Number Alterations With Survival in Metastatic Triple-Negative Breast Cancer”. J Clin Oncol 36: 543-53.
Integrative analysis reveals 53BP1 copy loss and decreased expression in a subset of human diffuse large B-cell lymphomas
Takeyama, K., S. Monti, J. P. Manis, P. Dal Cin, G. Getz, R. Beroukhim, S. Dutt, et al. 2008. “Integrative Analysis Reveals 53BP1 Copy Loss and Decreased Expression in a Subset of Human Diffuse Large B-Cell Lymphomas”. Oncogene 27: 318-22.
Large-scale chemical dissection of mitochondrial function
Wagner, B. K., T. Kitami, T. J. Gilbert, D. Peck, A. Ramanathan, S. L. Schreiber, T. R. Golub, and V. K. Mootha. 2008. “Large-Scale Chemical Dissection of Mitochondrial Function”. Nat Biotechnol 26: 343-51.
Chemical genomics identifies small-molecule MCL1 repressors and BCL-xL as a predictor of MCL1 dependency
Wei, G., A. A. Margolin, L. Haery, E. Brown, L. Cucolo, B. Julian, S. Shehata, A. L. Kung, R. Beroukhim, and T. R. Golub. 2012. “Chemical Genomics Identifies Small-Molecule MCL1 Repressors and BCL-XL As a Predictor of MCL1 Dependency”. Cancer Cell 21: 547-62.
Gene expression-based chemical genomics identifies rapamycin as a modulator of MCL1 and glucocorticoid resistance
Wei, G., D. Twomey, J. Lamb, K. Schlis, J. Agarwal, R. W. Stam, J. T. Opferman, et al. 2006. “Gene Expression-Based Chemical Genomics Identifies Rapamycin As a Modulator of MCL1 and Glucocorticoid Resistance”. Cancer Cell 10: 331-42.