A Re-Examination of Race in the U.S.
Wasswa, George. 2014. “A Re-Examination of Race in the U.S.”. Political Science 1 (15).
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Comprehensive assessment of cancer missense mutation clustering in protein structures
Kamburov, A., M. S. Lawrence, P. Polak, I. Leshchiner, K. Lage, T. R. Golub, E. S. Lander, and G. Getz. 2015. “Comprehensive Assessment of Cancer Missense Mutation Clustering in Protein Structures”. Proc Natl Acad Sci U S A 112: E5486-95.
Dicer1 functions as a haploinsufficient tumor suppressor
Kumar, M. S., R. E. Pester, C. Y. Chen, K. Lane, C. Chin, J. Lu, D. G. Kirsch, T. R. Golub, and T. Jacks. 2009. “Dicer1 Functions As a Haploinsufficient Tumor Suppressor”. Genes Dev 23: 2700-4.
A mechanism of cyclin D1 action encoded in the patterns of gene expression in human cancer
Lamb, J., S. Ramaswamy, H. L. Ford, B. Contreras, R. V. Martinez, F. S. Kittrell, C. A. Zahnow, N. Patterson, T. R. Golub, and M. E. Ewen. 2003. “A Mechanism of Cyclin D1 Action Encoded in the Patterns of Gene Expression in Human Cancer”. Cell 114: 323-34.
miR-126 Regulates Distinct Self-Renewal Outcomes in Normal and Malignant Hematopoietic Stem Cells
Lechman, E. R., B. Gentner, S. W. Ng, E. M. Schoof, P. Galen, J. A. Kennedy, S. Nucera, et al. 2016. “MiR-126 Regulates Distinct Self-Renewal Outcomes in Normal and Malignant Hematopoietic Stem Cells”. Cancer Cell 29: 214-28.
Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis
Levine, R. L., M. Wadleigh, J. Cools, B. L. Ebert, G. Wernig, B. J. Huntly, T. J. Boggon, et al. 2005. “Activating Mutation in the Tyrosine Kinase JAK2 in Polycythemia Vera, Essential Thrombocythemia, and Myeloid Metaplasia With Myelofibrosis”. Cancer Cell 7: 387-97.
A Library of Phosphoproteomic and Chromatin Signatures for Characterizing Cellular Responses to Drug Perturbations
Litichevskiy, L., R. Peckner, J. G. Abelin, J. K. Asiedu, A. L. Creech, J. F. Davis, D. Davison, et al. 2018. “A Library of Phosphoproteomic and Chromatin Signatures for Characterizing Cellular Responses to Drug Perturbations”. Cell Syst 6: 424-443 e7.
Incidence of TEL/AML1 fusion in children with relapsed acute lymphoblastic leukemia
Loh, M. L., L. B. Silverman, M. L. Young, D. Neuberg, T. R. Golub, S. E. Sallan, and D. G. Gilliland. 1998. “Incidence of TEL/AML1 Fusion in Children With Relapsed Acute Lymphoblastic Leukemia”. Blood 92: 4792-7.
Improved estimation of cancer dependencies from large-scale RNAi screens using model-based normalization and data integration
McFarland, J. M., Z. V. Ho, G. Kugener, J. M. Dempster, P. G. Montgomery, J. G. Bryan, J. M. Krill-Burger, et al. 2018. “Improved Estimation of Cancer Dependencies from Large-Scale RNAi Screens Using Model-Based Normalization and Data Integration”. Nat Commun 9: 4610.
TEL/AML-1 dimerizes and is associated with a favorable outcome in childhood acute lymphoblastic leukemia
McLean, T. W., S. Ringold, D. Neuberg, K. Stegmaier, R. Tantravahi, J. Ritz, H. P. Koeffler, et al. 1996. “TEL/AML-1 Dimerizes and Is Associated With a Favorable Outcome in Childhood Acute Lymphoblastic Leukemia”. Blood 88: 4252-8.