An ecosystem of cancer cell line factories to support a cancer dependency map
Boehm, J. S., and T. R. Golub. 2015. “An Ecosystem of Cancer Cell Line Factories to Support a Cancer Dependency Map”. Nat Rev Genet 16: 373-4.
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Identification of distinct molecular phenotypes in acute megakaryoblastic leukemia by gene expression profiling
Bourquin, J. P., A. Subramanian, C. Langebrake, D. Reinhardt, O. Bernard, P. Ballerini, A. Baruchel, et al. 2006. “Identification of Distinct Molecular Phenotypes in Acute Megakaryoblastic Leukemia by Gene Expression Profiling”. Proc Natl Acad Sci U S A 103: 3339-44.
Chemical genetic strategy identifies histone deacetylase 1 (HDAC1) and HDAC2 as therapeutic targets in sickle cell disease
Bradner, J. E., R. Mak, S. K. Tanguturi, R. Mazitschek, S. J. Haggarty, K. Ross, C. Y. Chang, et al. 2010. “Chemical Genetic Strategy Identifies Histone Deacetylase 1 (HDAC1) and HDAC2 As Therapeutic Targets in Sickle Cell Disease”. Proc Natl Acad Sci U S A 107: 12617-22.
Amplification of CRKL induces transformation and epidermal growth factor receptor inhibitor resistance in human non-small cell lung cancers
Cheung, H. W., J. Du, J. S. Boehm, F. He, B. A. Weir, X. Wang, M. Butaney, et al. 2011. “Amplification of CRKL Induces Transformation and Epidermal Growth Factor Receptor Inhibitor Resistance in Human Non-Small Cell Lung Cancers”. Cancer Discov 1: 608-25.
Expression analysis with oligonucleotide microarrays reveals that MYC regulates genes involved in growth, cell cycle, signaling, and adhesion
Coller, H. A., C. Grandori, P. Tamayo, T. Colbert, E. S. Lander, R. N. Eisenman, and T. R. Golub. 2000. “Expression Analysis With Oligonucleotide Microarrays Reveals That MYC Regulates Genes Involved in Growth, Cell Cycle, Signaling, and Adhesion”. Proc Natl Acad Sci U S A 97: 3260-5.
Genomic approaches to hematologic malignancies
Ebert, B. L., and T. R. Golub. 2004. “Genomic Approaches to Hematologic Malignancies”. Blood 104: 923-32.
NFkappaB activity, function, and target-gene signatures in primary mediastinal large B-cell lymphoma and diffuse large B-cell lymphoma subtypes
Feuerhake, F., J. L. Kutok, S. Monti, W. Chen, A. S. LaCasce, G. Cattoretti, P. Kurtin, et al. 2005. “NFkappaB Activity, Function, and Target-Gene Signatures in Primary Mediastinal Large B-Cell Lymphoma and Diffuse Large B-Cell Lymphoma Subtypes”. Blood 106: 1392-9.
TEL gene rearrangements in myeloid malignancy
Golub, T. R. 1997. “TEL Gene Rearrangements in Myeloid Malignancy”. Hematol Oncol Clin North Am 11: 1207-20.
Genomic approaches to the pathogenesis of hematologic malignancy
Golub, T. R. 2001. “Genomic Approaches to the Pathogenesis of Hematologic Malignancy”. Curr Opin Hematol 8: 252-61.
Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma
Green, M. R., S. Monti, S. J. Rodig, P. Juszczynski, T. Currie, E. O’Donnell, B. Chapuy, et al. 2010. “Integrative Analysis Reveals Selective 9p24.1 Amplification, Increased PD-1 Ligand Expression, and Further Induction via JAK2 in Nodular Sclerosing Hodgkin Lymphoma and Primary Mediastinal Large B-Cell Lymphoma”. Blood 116: 3268-77.