NMDA receptors in nervous system diseases.
Zhou, Qiang, and Morgan Sheng. 2013. “NMDA Receptors in Nervous System Diseases.”. Neuropharmacology 74: 69-75.
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Specific trans-synaptic interaction with inhibitory interneuronal neurexin underlies differential ability of neuroligins to induce functional inhibitory synapses.
Futai, Kensuke, Christopher D Doty, Brian Baek, Jubin Ryu, and Morgan Sheng. 2013. “Specific Trans-Synaptic Interaction With Inhibitory Interneuronal Neurexin Underlies Differential Ability of Neuroligins to Induce Functional Inhibitory Synapses.”. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 33 (8): 3612-23.
GKAP orchestrates activity-dependent postsynaptic protein remodeling and homeostatic scaling.
Shin, Seung Min, Nanyan Zhang, Jonathan Hansen, Nashaat Z Gerges, Daniel T S Pak, Morgan Sheng, and Sang H Lee. 2012. “GKAP Orchestrates Activity-Dependent Postsynaptic Protein Remodeling and Homeostatic Scaling.”. Nature Neuroscience 15 (12): 1655-66.
Synaptic structure and function.
Triller, Antoine, and Morgan Sheng. 2012. “Synaptic Structure and Function.”. Current Opinion in Neurobiology 22 (3): 363-5.
Leukocyte common antigen-related phosphatase is a functional receptor for chondroitin sulfate proteoglycan axon growth inhibitors.
Fisher, Daniel, Bin Xing, John Dill, Hui Li, Hai Hiep Hoang, Zhenze Zhao, Xiao-Li Yang, et al. 2011. “Leukocyte Common Antigen-Related Phosphatase Is a Functional Receptor for Chondroitin Sulfate Proteoglycan Axon Growth Inhibitors.”. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 31 (40): 14051-66.
Differential functional consequences of GRIN2A mutations associated with schizophrenia and neurodevelopmental disorders
Shepard, Nate, David Baez-Nieto, Sumaiya Iqbal, Erkin Kurganov, Nikita Budnik, Arthur J. Campbell, Jen Q. Pan, Morgan H Sheng, and Zohreh Farsi. 2024. “Differential Functional Consequences Of GRIN2A mutations Associated With Schizophrenia and Neurodevelopmental Disorders”. Scientific Reports .
Elevated synaptic PKA activity and abnormal striatal dopamine signaling in Akap11 mutant mice, a genetic model of schizophrenia and bipolar disorder
Song, Bryan, Yang Ge, Ally Nicolella, Min Jee Kwon, Bart Lodder, Kevin Bonanno, Antia Valle-Tojeiro, et al. 2025. “Elevated Synaptic PKA Activity and Abnormal Striatal Dopamine Signaling In Akap11 mutant Mice, a Genetic Model of Schizophrenia and Bipolar Disorder”. Nature Communications .
Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer’s disease mouse models
Dejanovic, Borislav, Tiffany Wu, Ming-Chi Tsai, David Graykowski, Vineela D. Gandham, Christopher M. Rose, Corey E. Bakalarski, et al. 2022. “Complement C1q-Dependent Excitatory and Inhibitory Synapse Elimination by Astrocytes and Microglia in Alzheimer’s Disease Mouse Models”. Nature Aging.
Targeting synapse function and loss for treatment of neurodegenerative diseases
Dejanovic, Borislav, Morgan H Sheng, and Jesse E. Hanson. 2024. “Targeting Synapse Function and Loss for Treatment of Neurodegenerative Diseases”. Nature Reviews Drug Discovery.
Performance across different versions of an artificial intelligence model for screen-reading of mammograms.
Larsen, Marthe, Christoph I Lee, Marie B Bergan, Åsne S Holen, Håkon Lund-Hanssen, Solveig R Hoff, Steinar Auensen, et al. 2026. “Performance across Different Versions of an Artificial Intelligence Model for Screen-Reading of Mammograms.”. European Radiology.