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Ines Picard
she/her
Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.
Wu, Tiffany, Borislav Dejanovic, Vineela D Gandham, Alvin Gogineni, Rose Edmonds, Stephen Schauer, Karpagam Srinivasan, et al. 2019. “Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.”. Cell Reports 28 (8): 2111-2123.e6.
Positive Allosteric Modulators of GluN2A-Containing NMDARs with Distinct Modes of Action and Impacts on Circuit Function.
Hackos, David H, Patrick J Lupardus, Teddy Grand, Yelin Chen, Tzu-Ming Wang, Paul Reynen, Amy Gustafson, et al. 2016. “Positive Allosteric Modulators of GluN2A-Containing NMDARs With Distinct Modes of Action and Impacts on Circuit Function.”. Neuron 89 (5): 983-99.
Local pruning of dendrites and spines by caspase-3-dependent and proteasome-limited mechanisms.
Ertürk, Ali, Yuanyuan Wang, and Morgan Sheng. 2014. “Local Pruning of Dendrites and Spines by Caspase-3-Dependent and Proteasome-Limited Mechanisms.”. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 34 (5): 1672-88.
Long-term depression: a cell biological view.
Sheng, Morgan, and Ali Ertürk. 2014. “Long-Term Depression: A Cell Biological View.”. Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences 369 (1633): 20130138.
Caspases in synaptic plasticity.
Li, Zheng, and Morgan Sheng. 2012. “Caspases in Synaptic Plasticity.”. Molecular Brain 5: 15.
Sheng Lab
Kyla Jacobs
she/her
Activity-induced Polo-like kinase 2 is required for homeostatic plasticity of hippocampal neurons during epileptiform activity.
Seeburg, Daniel P, and Morgan Sheng. 2008. “Activity-Induced Polo-Like Kinase 2 Is Required for Homeostatic Plasticity of Hippocampal Neurons During Epileptiform Activity.”. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 28 (26): 6583-91.