Dr. Wellington Hsu: Why robotic technology could revolutionize spine surgery, and where AI fits in News: February 22, 2020 Headline
Endogenous Gαq-Coupled Neuromodulator Receptors Activate Protein Kinase A. Publication: Journal Article Chen, Yao, Adam J Granger, Trinh Tran, Jessica L Saulnier, Alfredo Kirkwood, and Bernardo L Sabatini. 2017. “Endogenous Gαq-Coupled Neuromodulator Receptors Activate Protein Kinase A.”. Neuron 96 (5): 1070-1083.e5.
Corelease of acetylcholine and GABA from cholinergic forebrain neurons. Publication: Journal Article Saunders, Arpiar, Adam J Granger, and Bernardo L Sabatini. 2015. “Corelease of Acetylcholine and GABA from Cholinergic Forebrain Neurons.”. ELife 4.
CRISPR/Cas9-mediated gene knock-down in post-mitotic neurons. Publication: Journal Article Straub, Christoph, Adam J Granger, Jessica L Saulnier, and Bernardo L Sabatini. 2014. “CRISPR/Cas9-Mediated Gene Knock-down in Post-Mitotic Neurons.”. PloS One 9 (8): e105584.
Expression mechanisms underlying long-term potentiation: a postsynaptic view, 10 years on. Publication: Journal Article Granger, Adam J, and Roger A Nicoll. 2014. “Expression Mechanisms Underlying Long-Term Potentiation: A Postsynaptic View, 10 Years On.”. Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences 369 (1633): 20130136.
LTP requires a reserve pool of glutamate receptors independent of subunit type. Publication: Journal Article Granger, Adam J, Yun Shi, Wei Lu, Manuel Cerpas, and Roger A Nicoll. 2013. “LTP Requires a Reserve Pool of Glutamate Receptors Independent of Subunit Type.”. Nature 493 (7433): 495-500.
LTD expression is independent of glutamate receptor subtype. Publication: Journal Article Granger, Adam J, and Roger A Nicoll. 2014. “LTD Expression Is Independent of Glutamate Receptor Subtype.”. Frontiers in Synaptic Neuroscience 6: 15.
Genetic analysis of neuronal ionotropic glutamate receptor subunits. Publication: Journal Article Granger, Adam J, John A Gray, Wei Lu, and Roger A Nicoll. 2011. “Genetic Analysis of Neuronal Ionotropic Glutamate Receptor Subunits.”. The Journal of Physiology 589 (17): 4095-101.
Potentiation of synaptic AMPA receptors induced by the deletion of NMDA receptors requires the GluA2 subunit. Publication: Journal Article Lu, Wei, John A Gray, Adam J Granger, Matthew J During, and Roger A Nicoll. 2011. “Potentiation of Synaptic AMPA Receptors Induced by the Deletion of NMDA Receptors Requires the GluA2 Subunit.”. Journal of Neurophysiology 105 (2): 923-8.