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Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.
Wu, Tiffany, Borislav Dejanovic, Vineela D Gandham, Alvin Gogineni, Rose Edmonds, Stephen Schauer, Karpagam Srinivasan, et al. 2019. “Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.”. Cell Reports 28 (8): 2111-2123.e6.
Positive Allosteric Modulators of GluN2A-Containing NMDARs with Distinct Modes of Action and Impacts on Circuit Function.
Hackos, David H, Patrick J Lupardus, Teddy Grand, Yelin Chen, Tzu-Ming Wang, Paul Reynen, Amy Gustafson, et al. 2016. “Positive Allosteric Modulators of GluN2A-Containing NMDARs With Distinct Modes of Action and Impacts on Circuit Function.”. Neuron 89 (5): 983-99.
Local pruning of dendrites and spines by caspase-3-dependent and proteasome-limited mechanisms.
Ertürk, Ali, Yuanyuan Wang, and Morgan Sheng. 2014. “Local Pruning of Dendrites and Spines by Caspase-3-Dependent and Proteasome-Limited Mechanisms.”. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 34 (5): 1672-88.
Long-term depression: a cell biological view.
Sheng, Morgan, and Ali Ertürk. 2014. “Long-Term Depression: A Cell Biological View.”. Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences 369 (1633): 20130138.
Caspases in synaptic plasticity.
Li, Zheng, and Morgan Sheng. 2012. “Caspases in Synaptic Plasticity.”. Molecular Brain 5: 15.
Publications
https://www.ncbi.nlm.nih.gov/myncbi/seungmin.hwang.1/bibliography/public/
Publications
Sheng Lab
Vijay Sankaran, M.D., Ph.D.
Jan Ellen Paradise, MD Professor of Pediatrics, Harvard Medical School Investigator, Howard Hughes Medical Institute Attending Physician, Dana-Farber/Boston Children's Cancer and Blood Disorders Center